Abdul, M. ., Sultana, R. ., Keller, J. ., St Clair, D. ., Markesbery, W. ., & Butterfield, D. . (2006). Mutations in amyloid precursor protein and presenilin-1 genes increase the basal oxidative stress in murine neuronal cells and lead to increased sensitivity to oxidative stress mediated by amyloid beta-peptide (1-42), HO and kainic acid: implications.. Journal of Neurochemistry, 96(5), 1322-35. https://doi.org/10.1111/j.1471-4159.2005.03647.x

Abdul, M. ., & Butterfield, D. . (2005). Protection against amyloid beta-peptide (1-42)-induced loss of phospholipid asymmetry in synaptosomal membranes by tricyclodecan-9-xanthogenate (D609) and ferulic acid ethyl ester: implications for Alzheimer’s disease. Biochimica Et Biophysica Acta, 1741(1-2), 140-8. https://doi.org/10.1016/j.bbadis.2004.12.002 (Original work published 2005)

Boyd-Kimball, D. ., Abdul, M. ., Reed, T. ., Sultana, R. ., & Butterfield, D. . (2004). Role of phenylalanine 20 in Alzheimer’s amyloid beta-peptide (1-42)-induced oxidative stress and neurotoxicity. Chemical Research in Toxicology, 17(12), 1743-9. https://doi.org/10.1021/tx049796w

Abdul, M. ., Wenk, G. ., Gramling, M. ., Hauss-Wegrzyniak, B. ., & Butterfield, D. . (2004). APP and PS-1 mutations induce brain oxidative stress independent of dietary cholesterol: implications for Alzheimer’s disease. Neuroscience Letters, 368(2), 148-50. https://doi.org/10.1016/j.neulet.2004.06.077 (Original work published 2004)